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babouflo201223

Experienced
Aug 18, 2024
271
In case if full suspension, if everyting is ok, how much time does it need to die if nobody finds you ? And how happens the death ? Stopping heart ?
 
vanillamilkshakes

vanillamilkshakes

Aspiring Corpse
Aug 26, 2024
435
You may find these threads helpful:
 
Sunü (素女)

Sunü (素女)

No, I'm not chinese.
Sep 30, 2023
11
If you're aiming to achieve a hangman's fracture, I think death will be quite instant. Otherwise, it's death by strangulation (or if improperly setup, very painful blood loss), and I remember the estimate being around ~30 minutes of no interruptions to ensure no survivabiity (but the video below says our ancestors kept it up to an hour). Here's a Youtube video informing the science behind it:
 
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babouflo201223

Experienced
Aug 18, 2024
271
Ces fils de discussion peuvent vous être utiles :
Thank you, but these threads are about partial suspension (or I'm dumb...) and my question is about full suspension. But thank you !
If you're aiming to achieve a hangman's fracture, I think death will be quite instant. Otherwise, it's death by strangulation (or if improperly setup, very painful blood loss), and I remember the estimate being around ~30 minutes of no interruptions to ensure no survivabiity (but the video below says our ancestors kept it up to an hour). Here's a Youtube video informing the science behind it:

Thank you, but unfortunately hangman's fracture is a myth about quite instant death. It's just that if neck is broken, you can't move your body from toes to neck and you seem death but in fact you're not. Time to reach death remains the same.
But it' interesting when you say "if improperly setup, very painful blood loss". It's the first time I read that and of course I don't want it could happen. Blood loss from where ? Could you please tell me ? Thank you very much.
 
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ctbsd24

Member
Oct 8, 2024
89
From what I understand about the short drop method, if it's done correctly then what ultimately kills someone is cerebral hypoxia, i.e., lack of oxygen to the brain. Basically, the carotid arteries carry oxygen-rich blood to the brain. The noose closes off the arteries so no blood flows to the brain. This causes the person to pass out in a few seconds and, without being rescued, brain death likely occurs somewhere between 5 and 30 minutes later depending on the placement of the noose, the person's anatomy, etc.

It's my chosen method because while it takes a while to fully die, I'll be unconscious quickly and no longer aware of the time passage. I'll get to finally rest while gravity does its thing.
 
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L'absent

L'absent

À ma manière 🪦
Aug 18, 2024
801
In complete hanging, death occurs due to the dislocation of the atlanto-epistropheal joint and subsequent compression of the medulla oblongata by the tooth of the axis.

Hanging produces an immediate loss of consciousness and rapid death even when only one part of the body is hanging on the noose, therefore, finding individuals with a rope tightened around their neck standing, sitting or semi-reclining does not exclude this mode of death.

To occlude the airways, simply exert a traction equal to 1/3 of your body weight on the strap, while a traction of 3-4 kg is sufficient to interrupt the circulation of the carotid arteries and a simple compression of the vagus nerve and the carotid sinus receptors. it can produce immediate arrest of the heart with syncopal death due to reflex inhibition.

Se vogliamo essere più specifici, non è possibile rispondere alla tua domanda con una risposta chiara. Si può morire in vari modi per impiccagione e non si può sapere come è successo senza un'autopsia.

La morte per impiccagione è attribuibile a un fattore asfittico, a un fattore circolatorio ea un fattore neurovegetativo.



Fattore asfissiante: la cinghia, posta generalmente nello spazio tiroioideo, sposta all'indietro e verso l'alto l'osso ioide e la base della lingua che, premendo contro il palato e la faringe, determina l'occlusione delle vie aeree. Solo se interviene il fattore asfissiante possono verificarsi convulsioni asfittiche terminali.

Fattore circolatorio: l'interruzione della circolazione sanguigna a livello delle arterie carotidi situate ai lati del collo (3,5 kg) ed eventualmente delle arterie vertebrali (16,6 kg) producono ischemia cerebrale con immediata perdita di coscienza. La chiusura delle arterie giugulari determina invece una stasi venosa acuta nel territorio cefalico che produce edema.

Fattore neurovegetativo: una comprensione intensa del nervo vago, che decorre verticalmente nel fascio vascolo-nervoso del collo insieme all'arteria carotide anteriormente e alla vena giugulare posteriormente, e dei recettori del seno carotideo, può produrre l'arresto immediato del cuore con morte per inibizione riflessa.

On medical-legal examination in a hanging victim, in addition to the sulcus, there are: hemorrhages in the dermis, subcutaneous tissue, interstitial connective tissue and cervical muscles; tearing of the neck muscle fibers; fracture and dislocation of the hyoid bone; transverse rupture of the intima of the common carotid near its bifurcation (Amussat s.); ecchymosis in the adventitia of the carotids (Friedberg's syndrome); laceration of the nerve fibers of the vagus (S. di Dotto); retropharyngeal or prevertebral ecchymosis (Brouardel's disease); hemorrhages under the anterior longitudinal ligament of the vertebral column at the dorsal-lumbar passage (Simon's s.); intense cyanosis of the face or paleness; presence of hypostasis in the distal segments of the limbs and in the regions of the pelvis (pants hypostasis), which can cause erection of the penis with emission of sperm; punctate conjunctival hemorrhages and acute emphysema of the lungs.
The jump and the closing of the noose cause another very serious injury, the fracture of the cervical vertebra, but not SUFFICIENT to cause DEATH. Whether there is suffering and pain, it is not known. Don't believe anyone who tells you about immediate death with a broken neck, they are lies. Immediate death can only occur if you throw yourself from a point high enough to cause an improper decapitation.
 
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babouflo201223

Experienced
Aug 18, 2024
271
Tha
D'après ce que je comprends de la méthode de la chute courte, si elle est effectuée correctement, ce qui tue finalement quelqu'un est l'hypoxie cérébrale, c'est-à-dire le manque d'oxygène au cerveau. Fondamentalement, les artères carotides transportent le sang riche en oxygène vers le cerveau. Le nœud coulant ferme les artères, de sorte que le sang ne circule pas vers le cerveau. Cela provoque l'évanouissement de la personne en quelques secondes et, sans secours, la mort cérébrale survient probablement entre 5 et 30 minutes plus tard, selon l'emplacement du nœud coulant, l'anatomie de la personne, etc.

C'est ma méthode choisie car même si cela prend un certain temps pour mourir complètement, je serai rapidement inconscient et je ne serai plus conscient du passage du temps. Je pourrai enfin me reposer pendant que la gravité fait son travail.
Merci beaucoup pour vos explications. J'aimerais être aussi optimiste que vous mais malheureusement j'ai échoué ce soir again à la suspension totale du CTB. Le SI est tellement fort, même en prenant des pilules de benzodiazépines, et ce soir je n'ai même pas réussi à tomber de la chaise. Désespoir, car la suspension totale est la voie que je veux
In complete hanging, death occurs due to the dislocation of the atlanto-epistropheal joint and subsequent compression of the medulla oblongata by the tooth of the axis.

Hanging produces an immediate loss of consciousness and rapid death even when only one part of the body is hanging on the noose, therefore, finding individuals with a rope tightened around their neck standing, sitting or semi-reclining does not exclude this mode of death.

To occlude the airways, simply exert a traction equal to 1/3 of your body weight on the strap, while a traction of 3-4 kg is sufficient to interrupt the circulation of the carotid arteries and a simple compression of the vagus nerve and the carotid sinus receptors. it can produce immediate arrest of the heart with syncopal death due to reflex inhibition.

Se vogliamo essere più specifici, non è possibile rispondere alla tua domanda con una risposta chiara. Si può morire in vari modi per impiccagione e non si può sapere come è successo senza un'autopsia.

La morte per impiccagione è attribuibile a un fattore asfittico, a un fattore circolatorio ea un fattore neurovegetativo.



Fattore asfissiante: la cinghia, posta generalmente nello spazio tiroioideo, sposta all'indietro e verso l'alto l'osso ioide e la base della lingua che, premendo contro il palato e la faringe, determina l'occlusione delle vie aeree. Solo se interviene il fattore asfissiante possono verificarsi convulsioni asfittiche terminali.

Fattore circolatorio: l'interruzione della circolazione sanguigna a livello delle arterie carotidi situate ai lati del collo (3,5 kg) ed eventualmente delle arterie vertebrali (16,6 kg) producono ischemia cerebrale con immediata perdita di coscienza. La chiusura delle arterie giugulari determina invece una stasi venosa acuta nel territorio cefalico che produce edema.

Fattore neurovegetativo: una comprensione intensa del nervo vago, che decorre verticalmente nel fascio vascolo-nervoso del collo insieme all'arteria carotide anteriormente e alla vena giugulare posteriormente, e dei recettori del seno carotideo, può produrre l'arresto immediato del cuore con morte per inibizione riflessa.

Français À l'examen médico-légal d'une victime pendue, on observe, outre le sulcus, : des hémorragies du derme, du tissu sous-cutané, du tissu conjonctif interstitiel et des muscles cervicaux ; une déchirure des fibres musculaires du cou ; une fracture et une luxation de l'os hyoïde ; une rupture transversale de l'intima de la carotide commune près de sa bifurcation (syndrome d'Amussat) ; des ecchymoses dans l'adventice des carotides (syndrome de Friedberg) ; une lacération des fibres nerveuses du vague (syndrome de S. di Dotto) ; des ecchymoses rétropharyngées ou prévertébrales (maladie de Brouardel) ; des hémorragies sous le ligament longitudinal antérieur de la colonne vertébrale au niveau du passage dorso-lombaire (syndrome de Simon) ; une cyanose intense du visage ou une pâleur ; présence d'hypostase dans les segments distaux des membres et dans les régions du bassin (hypostase du pantalon), pouvant provoquer une érection du pénis avec émission de sperme; hémorragies conjonctivales ponctuées et emphysème aigu des poumons.
Le saut et la fermeture du nœud coulant provoquent une autre blessure très grave, la fracture de la vertèbre cervicale, mais pas SUFFISANTE pour provoquer la MORT. On ne sait pas s'il y a souffrance et douleur. Ne croyez pas ceux qui vous parlent d'une mort immédiate avec une fracture du cou, ce sont des mensonges. La mort immédiate ne peut survenir que si vous vous jetez d'un point suffisamment haut pour provoquer une décapitation injustifiée.
Thank you very much for your detailed explanations.
 
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babouflo201223

Experienced
Aug 18, 2024
271
D'après ce que je comprends de la méthode de la chute courte, si elle est effectuée correctement, ce qui tue finalement quelqu'un est l'hypoxie cérébrale, c'est-à-dire le manque d'oxygène au cerveau. Fondamentalement, les artères carotides transportent le sang riche en oxygène vers le cerveau. Le nœud coulant ferme les artères, de sorte que le sang ne circule pas vers le cerveau. Cela provoque l'évanouissement de la personne en quelques secondes et, sans secours, la mort cérébrale survient probablement entre 5 et 30 minutes plus tard, selon l'emplacement du nœud coulant, l'anatomie de la personne, etc.

C'est ma méthode choisie car même si cela prend un certain temps pour mourir complètement, je serai rapidement inconscient et je ne serai plus conscient du passage du temps. Je pourrai enfin me reposer pendant que la gravité fait son travail.
A question : when you fall into unconsciousness (full suspension), are we 100% sure that there is no risk to wake up before death happens (even if nobody comes to save you) ?
 
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ctbsd24

Member
Oct 8, 2024
89
A question : when you fall into unconsciousness (full suspension), are we 100% sure that there is no risk to wake up before death happens (even if nobody comes to save you) ?
My knee jerk reaction is no, though I'm sure someone on here could provide examples of folks who woke up. I just don't see how it's possible if the carotids remain sealed off the entire time. I hope that's true anyway lol. As soon as I muster the courage to step off my chair, I want that to be it. I have no intention of waking up after the lights go out in my head.
 
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